Respan Dataset Explorer
Select one behavior. Every returned turn has one binary label: Present or Absent. Source: final dense boolean release.
5,167,182physical rows
86shards
0.00%qualified row coverage
0.00%qualified cell coverage
turns-00019.parquet:71586
54ad5b8d9035ee9bdd6ec099
turn 10/19gpt-3.5-turbo-0125EnglishBelarus24 words
degenerate_repetitionAbsentFinal dense release
USER
Choose the main source of energy for red blood cells in fasting and starvation:
Select one:
Fatty acids
Ketone bodies
Glucose
Amino acids
turns-00019.parquet:71587
3577c2dd826d1609ecda2eef
turn 11/19gpt-3.5-turbo-0125EnglishBelarus17 words
degenerate_repetitionAbsentFinal dense release
USER
For the 1st week of starvation the following points are characteristic:
Hyperlipoproteinemia
Azotemia
Ketonemia
Hypoglucosemia
Acidosis
turns-00019.parquet:71588
9d3b0bf9bd8a64697c47c860
turn 12/19gpt-3.5-turbo-0125EnglishBelarus75 words
degenerate_repetitionAbsentFinal dense release
USER
Choose the correct statement. After 3 weeks of starvation:
Select one:
Enzymes of gluconeogenesis and ketogenesis are inactivated in the liver
The source of energy in hepatocytes is glucose
The level of ketone bodies is almost 100-fold increased, there is a threat for the development of metabolic acidosis
Neurons use only glucose as asource of energy
ASSISTANT
The level of ketone bodies is almost 100-fold increased, there is a threat for the development of metabolic acidosis
turns-00019.parquet:71589
28d24fe02354e9e62ae955e5
turn 13/19gpt-3.5-turbo-0125EnglishBelarus21 words
degenerate_repetitionAbsentFinal dense release
USER
All following substrates can be used for gluconeogenesis during fasting, except:
Select one:
Glycerol
Arachidonic acid
Alanine
Glutamic acid
ASSISTANT
Arachidonic acid
turns-00019.parquet:71590
2283ee1e26019e24f7d6741f
turn 14/19gpt-3.5-turbo-0125EnglishBelarus39 words
degenerate_repetitionAbsentFinal dense release
USER
In starvation during one week the blood glucose level is maintained due to:
Gluconeogenesis from glycerol
Glycogen mobilization from the liver
Gluconeogenesis from lactate
Decreased influx of glucose to muscles
Synthesis of glucose from amino acids
ASSISTANT
Gluconeogenesis from lactate
turns-00019.parquet:71591
2c1d1ab0ce92ffafee4e39b2
turn 15/19gpt-3.5-turbo-0125EnglishBelarus67 words
degenerate_repetitionAbsentFinal dense release
USER
All statements regarding organ specialization of metabolism after a meal are true, except:
Select one:
Glucose in red blood cells is oxidized to lactate
The main sources of energy in muscles are fatty acids
Glucose in muscles is used for the formation of glycogen
Ketone bodies are extensively used as a source of energy in the brain
ASSISTANT
The main sources of energy in muscles are fatty acids
turns-00019.parquet:71592
de729562686da44859813f74
turn 16/19gpt-3.5-turbo-0125EnglishBelarus30 words
degenerate_repetitionAbsentFinal dense release
USER
The main source of blood glucose during overnight fasting is:
Select one:
Glycogenolysis in muscles
Gluconeogenesis
Transfer of glucose from the intestine
Glycogenolysis in the liver
ASSISTANT
Glycogenolysis in the liver
turns-00019.parquet:71593
307565451458047d88f73f46
turn 17/19gpt-3.5-turbo-0125EnglishBelarus28 words
degenerate_repetitionAbsentFinal dense release
USER
Approximately, 1.5 hours after eating well- balanced food the levels of all following compounds are increased in blood, except:
Select one:
Chylomicrons
Fatty acids
Epinephrine
O Glucose
turns-00019.parquet:71594
c66d25240de5bcb1da9ddaca
turn 18/19gpt-3.5-turbo-0125EnglishBelarus21 words
degenerate_repetitionAbsentFinal dense release
USER
Glucose is the source of all these substrates for lipogenesis, except:
Select one:
Glycerol-3-phosphate
O Biotin
O Acetyl-CoA
O NADPH.H+
turns-00019.parquet:71595
0dafbcb3deaaafe8b32e29b9
turn 19/19gpt-3.5-turbo-0125EnglishBelarus47 words
degenerate_repetitionAbsentFinal dense release
USER
The cause of increased ketone bodies in blood in starvation is:
Acceleration of lipolysis in adipose tissue
☐ Decreased insulin/glucagon ratio
☐ Gluconeogenesis acceleration
Activation of HMG-CoA synthase in the liver mitochondria
☐ Acceleration of ẞ-oxidation in the liver
ASSISTANT
Activation of HMG-CoA synthase in the liver mitochondria